瘧疾自測試劑盒

廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發(fā)、銷售、服務于一體的優(yōu)良企業(yè),公司產(chǎn)品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家有名診斷產(chǎn)品集團公司產(chǎn)品，致力于為商檢單位、疾病預防控制中心、海關出入境檢疫局、衛(wèi)生防疫單位,緝毒系統(tǒng)，戒毒中心，檢驗檢疫單位、生化企業(yè)、科研院所、醫(yī)療機構等機構與行業(yè)提供*、高品質(zhì)的產(chǎn)品服務。此外，本公司還開展食品、衛(wèi)生、環(huán)境、藥品等多方面的第三方檢測服務。）

瘧疾自測試劑盒 本試劑盒主要是采用膠體金層析的原理制成，用于檢測人體血清/血漿/全血標本中，感染的瘧原蟲抗體，包括了惡性瘧原蟲和間日瘧原蟲、卵形瘧原蟲、三日瘧原蟲共有抗原的鑒別性檢測。

人群易感性 人群對瘧疾普遍易感，感染后雖有一定的免疫力，但不持久，各型瘧疾之間亦無交叉免疫性，經(jīng)反復多次感染后，再感染時癥狀可較輕，甚至無癥狀，而一般非流行區(qū)來的外來人員常較易感染，且癥狀較重。
People susceptible to the crowd generally susceptible to malaria, although the infection after a certain degree of immunity, but not lasting, there is no cross-immunity between malaria, after repeated infections, re-infection symptoms may be lighter, or even Asymptomatic, while the non-endemic areas of non-migrant workers are often more susceptible to infection, and the symptoms are severe.

：

1 撕開檢測卡鋁箔袋，取出袋內(nèi)金標卡。注意：不要讓袋內(nèi)材料暴露于高溫高濕環(huán)境，撕開鋁箔袋后盡快使用。

2將金標卡平放在臺面上；并將病人名字和編號寫在標簽上。

3 取5微升(吸管*刻度處)全血標本，垂直加入金標卡上“加樣孔A”內(nèi)。

4 掰斷裂解液瓶子蓋子上方的綠色圓頭，在“樣品孔B”上垂直滴加4滴裂解液。

5 在十五分鐘內(nèi)出結果。注意：必須在15分鐘內(nèi)判讀結果，如超時判斷，結果無效。

6 請遵循相關法規(guī)，妥善處理樣本及廢棄材料。

7 存儲條件：2-30℃；

8 保質(zhì)期：18個月；

【病原學檢測】

瘧疾檢測,用于檢測出虐疾的病原體——瘧原蟲，是明確診斷的zui直接證據(jù)。目前常用的層析法，具有操作簡單、靈敏度高和可鑒別蟲種等優(yōu)點，廣泛用于瘧疾的病原學診斷，是目前zui常用的方法之一。

我司為美國NOVABIOS公司在中國地區(qū)戰(zhàn)略合作伙伴，負責該公司產(chǎn)品的總經(jīng)銷及售后服務工作。還與各疾控中心，疾病防御中心有合作關系，例如中國疾病預防控制中心 、浙江省疾病預防控制中心  ，詳情可以我司工作人員。

（  MOB：楊永漢）  

我司還提供其它進口或國產(chǎn)試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產(chǎn)品。

廣州健侖生物長期供應各種違禁品檢測試紙、違禁品檢測卡、違禁品檢測試劑盒、藥篩試紙、藥篩試劑盒、嗎啡檢測試劑盒、巴比妥檢測試劑盒等。

想了解更多的產(chǎn)品及服務請掃描下方二維碼：

【公司名稱】 廣州健侖生物科技有限公司
【市場部】    楊永漢

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【騰訊  】 
【公司地址】 廣州清華科技園創(chuàng)新基地番禺石樓鎮(zhèn)創(chuàng)啟路63號二期2幢101-103

3.易感者人群
主要取決于接觸病原體的程度和頻率。青壯年因職業(yè)（農(nóng)民、牧民、獸醫(yī)、屠宰場和皮毛加工廠工人等）關系與病畜及其皮毛和排泄物、帶芽胞的塵埃等的接觸機會較多，其發(fā)病率也較高。zui為多見，可分炭疽癰和惡性水腫兩型。炭疽多見于面、頸、肩、手和腳等裸露部位皮膚，初為丘疹或斑疹，第2日頂部出現(xiàn)水皰，內(nèi)含淡黃色液體，周圍組織硬而腫，第3～4日中心區(qū)呈現(xiàn)出血性壞死，稍下陷，周圍有成群小水皰，水腫區(qū)繼續(xù)擴大。第5～7日水皰壞死破裂成淺小潰瘍，血樣分泌物結成黑色似炭塊的干痂，痂下有肉芽組織形成為炭疽癰。周圍組織有非凹陷性水腫。黑痂壞死區(qū)的直徑大小不等，自1～2cm至5～6cm，水腫區(qū)直徑可達5～20cm，堅實、疼痛不著、潰瘍不化膿等為其特點。繼之水腫漸退，黑痂在1～2周內(nèi)脫落，再過1～2周愈合成疤。發(fā)病1～2日后出現(xiàn)發(fā)熱、頭痛、局部淋巴結腫大及脾腫大等。
少數(shù)病例局部無黑痂形成而呈現(xiàn)大塊狀水腫，累及部位大多為組織疏松的眼瞼、頸、大腿等，患處腫脹透明而堅韌，擴展迅速，可致大片壞死。全身毒血癥明顯，病情危重，若治療貽誤，可因循環(huán)衰竭而死亡。如病原菌進入血液，可產(chǎn)生敗血癥，并繼發(fā)肺炎及腦膜炎。
2.肺炭疽
大多為原發(fā)性，由吸入炭疽桿菌芽胞所致，也可繼發(fā)于皮膚炭疽。起病多急驟，但一般先有2～4日的感冒樣癥狀，且在緩解后再突然起病，呈雙相型。臨床表現(xiàn)為寒戰(zhàn)、高熱、氣急、呼吸困難、喘鳴、發(fā)紺、血樣痰、胸痛等，有時在頸、細菌出現(xiàn)皮下水腫。肺部僅聞及散在的細濕啰音，或有腦膜炎體征，體征與病情嚴重程度常不成比例?；颊卟∏榇蠖辔Ｖ?，常并發(fā)敗血癥和感染性休克，偶也可繼發(fā)腦膜炎。若不及時診斷與搶救，則常在急性癥狀出現(xiàn)后24～48小時因呼吸、循環(huán)衰竭而死亡。
3.腸炭疽
可表現(xiàn)為急性胃腸炎型和急腹癥型。前者潛伏期12～18小時，同食者可同時或相繼出現(xiàn)嚴重嘔吐、腹痛、水樣腹瀉，多于數(shù)日內(nèi)迅速康復。后者起病急驟，有嚴重毒血癥癥狀、持續(xù)性嘔吐、腹瀉、血水樣便、腹脹、腹痛等，腹部有壓痛或呈腹膜炎征象，若不及時治療，常并發(fā)敗血癥和感染性休克而于起病后3～4日內(nèi)死亡。
3. susceptible people
Mainly depends on the degree and frequency of exposure to pathogens. Young adults are more likely to have access to sick animals, their fur and their excrement, spores with sprouts, etc. due to occupations (peasants, pastoralists, veterinarians, slaughterhouses and fur factory workers, etc.) and their incidence is also high. The most common, can be divided into two types of anthrax and malignant edema. Anthrax more common in the face, neck, shoulders, hands and feet and other exposed parts of the skin, the first papules or rash, the top of the 2nd day blisters, containing a light yellow liquid around the hard and swollen organizations, the first 3 to 4 Center Hemorrhagic necrosis, slightly sag, surrounded by small blisters, edema area continues to expand. On the 5th to 7th day, vesicular necrosis ruptured into shallow ulcers. The blood samples secreted black like carbon blocks, and the granulation tissue under the calluses became anthrax. Non-pitting edema around the organization. The diameter of the black callus necrosis area varies in size, from 1 ~ 2cm to 5 ~ 6cm, edema diameter up to 5 ~ 20cm, solid, pain, ulcers do not suppurate its characteristics. Followed by edema receded, black scab off in 1 to 2 weeks, and then 1 to 2 weeks into a scar. 1 to 2 days after the onset of fever, headache, local lymph nodes and splenomegaly.
A small number of cases of non-black scab formed locally and showed massive edema, mostly involving the loose tissue of the eyelids, neck, thighs, swelling of the affected area is transparent and tough, rapid expansion can cause large necrosis. Systemic toxemia obvious, critically ill, if the treatment of bungling, may be due to circulatory failure and death. Such as pathogenic bacteria into the blood can produce sepsis and secondary to pneumonia and meningitis.
2. lung anthrax
Mostly primary, caused by inhalation of Bacillus anthracis spores, but also secondary to skin anthrax. More rapid onset, but generally the first 2 to 4 days of cold-like symptoms, and then suddenly onset after relief, was bipolar. Clinical manifestations of chills, fever, shortness of breath, dyspnea, wheezing, cyanosis, blood sputum, chest pain, etc., sometimes in the neck, bacteria subcutaneous edema. The lungs are only heard and scattered in the wet rales, or signs of meningitis, signs and the severity of the disease is often disproportionate. Most patients are critically ill, often complicated by septicemia and septic shock, even secondary meningitis. If not timely diagnosis and rescue, often after the onset of acute symptoms of 24 to 48 hours due to respiratory failure, circulatory failure and death.
Intestinal anthrax
Can be expressed as acute gastroenteritis and acute abdomen type. The former incubation period of 12 to 18 hours, with the same time or eater may have severe vomiting, abdominal pain, watery diarrhea, more than a few days rapid recovery. The latter has a sharp onset, severe symptoms of sepsis, persistent vomiting, diarrhea, bloody stool, abdominal distension, abdominal pain, abdominal tenderness or signs of peritonitis, if not treated, often complicated by sepsis and septic shock 3 to 4 days after the onset of death